Document 0586
 DOCN  M9590586
 TI    [From IgE to the cell adhesion molecule]
 DT    9509
 AU    Stain JP; C.H.U. de Rouen.
 SO    Allerg Immunol (Paris). 1995 Apr;27(4):111-5. Unique Identifier :
       AIDSLINE MED/95290084
 AB    The aim of this work is to contribute to increasing our knowledge of
       asthma. The IgE can't alone be imputed in the anaphylactic reaction.
       Asthma is a bronchial inflammatory disease. The secretion of lymphokines
       increases the inflammatory response. The eosinophils cells damage the
       respiratory epithelium. The mast cells and the basophils cells release
       the chemical mediators and also the cytokines. The adhesion molecules
       situated in the cell membrane permit leukocyte cells to integrate with
       extracellular matrix during intra-tissue migration. Adhesion molecules
       may play a primary role in the pathogenesis of inflammatory response.
       The key role of the CD4 + T lymphocytes subset appeared in the last
       years.
 DE    Asthma/*PHYSIOPATHOLOGY  Bronchitis/PHYSIOPATHOLOGY  Cell Adhesion
       Molecules/*PHYSIOLOGY  Chemotaxis, Leukocyte  English Abstract
       Eosinophils/SECRETION  Extracellular Matrix/PHYSIOLOGY  Human
       IgE/*IMMUNOLOGY  Lymphokines/PHYSIOLOGY  Mast Cells/SECRETION  Th2
       Cells/IMMUNOLOGY  JOURNAL ARTICLE  REVIEW  REVIEW, TUTORIAL

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